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Ccdc3: A new P63 target involved in regulation of liver lipid metabolism
Liao,Wenjuan1; Liu,Hongbing1,6; Zhang,Yiwei1; Jung,Ji Hoon1; Chen,Jiaxiang1,7; Su,Xiaohua2; Kim,Yeong C.3; Flores,Elsa R.2; Wang,San Ming3,8; Czarny-Ratajczak,Malwina4; Li,Wen5; Zeng,Shelya X.1; Lu,Hua1
2017-12-01
Source PublicationScientific Reports
Volume7Issue:1
Abstract

TAp63, a member of the p53 family, has been shown to regulate energy metabolism. Here, we report coiled coil domain-containing 3 (CCDC3) as a new TAp63 target. TAp63, but not ΔNp63, p53 or p73, upregulates CCDC3 expression by directly binding to its enhancer region. The CCDC3 expression is markedly reduced in TAp63-null mouse embryonic fibroblasts and brown adipose tissues and by tumor necrosis factor alpha that reduces p63 transcriptional activity, but induced by metformin, an anti-diabetic drug that activates p63. Also, the expression of CCDC3 is positively correlated with TAp63 levels, but conversely with ΔNp63 levels, during adipocyte differentiation. Interestingly, CCDC3, as a secreted protein, targets liver cancer cells and increases long chain polyunsaturated fatty acids, but decreases ceramide in the cells. CCDC3 alleviates glucose intolerance, insulin resistance and steatosis formation in transgenic CCDC3 mice on high-fat diet (HFD) by reducing the expression of hepatic PPARγ and its target gene CIDEA as well as other genes involved in de novo lipogenesis. Similar results are reproduced by hepatic expression of ectopic CCDC3 in mice on HFD. Altogether, these results demonstrate that CCDC3 modulates liver lipid metabolism by inhibiting liver de novo lipogenesis as a downstream player of the p63 network.

DOI10.1038/s41598-017-09228-8
URLView the original
Language英語English
WOS IDWOS:000408107000154
Scopus ID2-s2.0-85027859714
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Document TypeJournal article
CollectionUniversity of Macau
Corresponding AuthorZeng,Shelya X.
Affiliation1.Department of Biochemistry and Molecular Biology,Tulane Cancer Center,Tulane University School of Medicine,New Orleans,70112,United States
2.Department of Molecular Oncology,Cancer Biology and Evolution Program,H. Lee Moffitt Cancer Center,Tampa,United States
3.Department of Genetics,Cell Biology and Anatomy,University of Nebraska Medical Center,986805 Nebraska Medical Center,Omaha,68198,United States
4.Department of Medicine,Center for Aging,Tulane University School of Medicine,New Orleans,70112,United States
5.Laboratory of General Surgery,First Affiliated Hospital,Sun Yatsen University,Guangzhou, Guangdong,510080,China
6.Department of Pediatrics,Tulane University,School of Medicine,New Orleans,70112,United States
7.Department of Physiology,Jiangxi Medical College,Nanchang University,Nanchang, Jiangxi,330006,China
8.Faculty of Health Sciences,University of Macau,Macau,Macao
Recommended Citation
GB/T 7714
Liao,Wenjuan,Liu,Hongbing,Zhang,Yiwei,et al. Ccdc3: A new P63 target involved in regulation of liver lipid metabolism[J]. Scientific Reports, 2017, 7(1).
APA Liao,Wenjuan., Liu,Hongbing., Zhang,Yiwei., Jung,Ji Hoon., Chen,Jiaxiang., Su,Xiaohua., Kim,Yeong C.., Flores,Elsa R.., Wang,San Ming., Czarny-Ratajczak,Malwina., Li,Wen., Zeng,Shelya X.., & Lu,Hua (2017). Ccdc3: A new P63 target involved in regulation of liver lipid metabolism. Scientific Reports, 7(1).
MLA Liao,Wenjuan,et al."Ccdc3: A new P63 target involved in regulation of liver lipid metabolism".Scientific Reports 7.1(2017).
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