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JMJD1C is required for the survival of acute myeloid leukemia by functioning as a coactivator for key transcription factors
Mo Chen1; Nan Zhu2,6; Xiaochuan Liu3; Benoit Laurent4,5; TANG ZIKANG1; Rowena Eng2; Yang Shi4,5; Scott A. Armstrong2; Robert G. Roeder1
2015-10
Source PublicationGenes & development
Document TypeJournal article
CollectionINSTITUTE OF APPLIED PHYSICS AND MATERIALS ENGINEERING
Affiliation1.Laboratory of Biochemistry and Molecular Biology, The Rockefeller University, New York, New York 10065, USA
2.Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center, New York, New York 10065, USA
3.Department of Microbiology, Biochemistry, and Molecular Genetics, Rutgers University, Newark, New Jersey 07103, USA
4.Division of Newborn Medicine, Epigenetics Program, Department of Medicine, Boston Children's Hospital, Boston, Massachusetts 02115, USA
5.Department of Cell Biology, Harvard Medical School, Boston, Massachusetts 02115, USA
6.Present address: Blood Research Institute, Blood Center of Wisconsin, Milwaukee, Wisconsin 53226, USA
Recommended Citation
GB/T 7714
Mo Chen,Nan Zhu,Xiaochuan Liu,et al. JMJD1C is required for the survival of acute myeloid leukemia by functioning as a coactivator for key transcription factors[J]. Genes & development, 2015.
APA Mo Chen., Nan Zhu., Xiaochuan Liu., Benoit Laurent., TANG ZIKANG., Rowena Eng., Yang Shi., Scott A. Armstrong., & Robert G. Roeder (2015). JMJD1C is required for the survival of acute myeloid leukemia by functioning as a coactivator for key transcription factors. Genes & development.
MLA Mo Chen,et al."JMJD1C is required for the survival of acute myeloid leukemia by functioning as a coactivator for key transcription factors".Genes & development (2015).
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